Aryl hydrocarbon receptor repressor (AhR repressor) (AhRR) (Class E basic helix-loop-helix protein 77) (bHLHe77)
1_MPRTM 6_ IPPGE 11_ CTYAG 16_ RKRRR 21_ PLQKQ 26_ RPAVG 31_ AEKSN 36_ PSKRH 41_ RDRLN 46_ AELDH 51_ LASLL 56_ PFPPD 61_ IISKL 66_ DKLSV 71_ LRLSV 76_ SYLRV 81_ KSFFQ 86_ VVQEQ 91_ SSRQP 96_ AAGAP 101_ SPGDS 106_ CPLAG 111_ SAVLE 116_ GRLLL 121_ ESLNG 126_ FALVV 131_ SAEGT 136_ IFYAS 141_ ATIVD 146_ YLGFH 151_ QTDVM 156_ HQNIY 161_ DYIHV 166_ DDRQD 171_ FCRQL 176_ HWAMD 181_ PPQVV 186_ FGQPP 191_ PLETG 196_ DDAIL 201_ GRLLR 206_ AQEWG 211_ TGTPT 216_ EYSAF 221_ LTRCF 226_ ICRVR 231_ CLLDS 236_ TSGFL 241_ TMQFQ 246_ GKLKF 251_ LFGQK 256_ KKAPS 261_ GAMLP 266_ PRLSL 271_ FCIAA 276_ PVLLP 281_ SAAEM 286_ KMRSA 291_ LLRAK 296_ PRADT 301_ AATAD 306_ AKVKA 311_ TTSLC 316_ ESELH 321_ GKPNY 326_ SAGRS 331_ SRESG 336_ VLVLR 341_ EQTDA 346_ GRWAQ 351_ VPARA 356_ PCLCL 361_ RGGPD 366_ LVLDP 371_ KGGSG 376_ DREEE 381_ QHRML 386_ SRASG 391_ VTGRR 396_ ETPGP 401_ TKPLP 406_ WTAGK 411_ HSEDG 416_ ARPRL 421_ QPSKN 426_ DPPSL 431_ RPMPR 436_ GSCLP 441_ CPCVQ 446_ GTFRN 451_ SPISH 456_ PPSPS 461_ PSAYS 466_ SRTSR 471_ PMRDV 476_ GEDQV 481_ HPPLC 486_ HFPQR 491_ SLQHQ 496_ LPQPG 501_ AQRFA 506_ TRGYP 511_ MEDMK 516_ LQGVP 521_ MPPGD 526_ LCGPT 531_ LLLDV 536_ SIKME 541_ KDSGC 546_ EGAAD 551_ GCVPS 556_ QVWLG 561_ ASDRS 566_ HPATF 571_ PTRMH 576_ LKTEP 581_ DSRQQ 586_ VYISH 591_ LGHGV 596_ RGAQP 601_ HGRAT 606_ AGRSR 611_ ELTPF 616_ HPAHC 621_ ACLEP 626_ TDGLP 631_ QSEPP 636_ HQLCA 641_ RGRGE 646_ QSCTC 651_ RAAEA 656_ APVVK 661_ REPLD 666_ SPQWA 671_ THSQG 676_ MVPGM 681_ LPKSA 686_ LATLV 691_ PPQAS 696_GCTFL
1: Mediates dioxin toxicity and is involved in regulation of cell growth and differentiation. Represses the transcription activity of AHR by competing with this transcription factor for heterodimer formation with the ARNT and subsequently binding to the xenobiotic response element (XRE) sequence present in the promoter regulatory region of variety of genes. Represses CYP1A1 by binding the XRE sequence and recruiting ANKRA2, HDAC4 and/or HDAC5. Autoregulates its expression by associating with its own XRE site